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PostPosted: Fri Mar 23, 2007 8:34 am Reply with quoteBack to top

Animated Fractal Noise, Displacing a simple plane.
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Buy flagyl 750 mg /kg. In the first year after drug was approved, a single dose daily use was associated with increased levels of serum testosterone (p < 0.01, Figure ). We analyzed all trials and found Flagyl er $0.6 - pills Per pill a consistent evidence of increased circulating DHT over time ( Figure ). The median time between starting dose and serum testosterone peak was 8–16 hours. Although there were a few studies that did not report serum testosterone levels, the median time from first treatment event to the testosterone peak was 12 hours. Mean testosterone values were 10% higher at treatment initiation compared with the final time point of treatment. The median mean increase in serum testosterone during treatment was 2%, indicating the degree to which peak of serum DHT is reached in men taking a single dose of flagyl compared with men taking multiple doses. Discussion In this multicentre, double-blind, placebo-controlled, parallel-group study our data show that, even though testosterone supplementation was associated with suppression of endogenous testosterone production, levels in men who received treatment over a median time of 12 hours were significantly higher at the last treatment event compared with men who received treatment over a mean time of only 8 hours. Further, the peak time of serum DHT was reached almost immediately in men receiving treatment over a mean interval of 12 hours compared with 7 in men given placebo. Despite these encouraging results, it is still possible that a lower peak time for DHT was reached in men receiving treatment over a mean interval of 12 hours compared with men given placebo. This is supported by the finding that a lower maximum increase in mean serum DHT during treatment was reached in men receiving treatment over a median time of 12 hours compared with men given placebo. This finding is intriguing. As testosterone has both an androgenic and androgen non-specific action [18, 19], the discrepancy between findings in this study and those of others supporting a DHT peak time in the early morning or evening [7, 8] is not surprising. We hypothesized that, as the DHT level declined with time, the levels of circulating testosterone would remain elevated and thus lead to an observed difference in serum DHT levels between treatment groups. This has been shown previously in some studies of long-term DHT suppression [36]. In the present study, it would appear that levels of an androgenic hormone were higher than expected after a long-term treatment, causing the elevated serum DHT levels seen in men who received treatment over a long time. The present study did find that treatment with testosterone caused a significant dose-dependent suppression of endogenous estradiol levels. We could not see a significant difference in levels of DHT between those receiving treatment for 12 hours or 7 hours. The difference in DHT levels we found was statistically indistinguishable from a placebo effect. The reason for this discrepancy could be explained by a number of theoretical mechanisms. One possibility is that the change in DHT levels with time is a result of changes in the hormone biosynthetic pathway. A related possibility is that changes in the endogenous steroid biosynthetic pathway are a result of change in plasma levels testosterone. Although most of our data supports the hypothesis that changes in endogenous steroid biosynthetic pathways, rather than changes in plasma levels of testosterone, were involved in the observed suppression of endogenous estradiol production (see below), it is not clear how long the DHT and estradiol levels were suppressed by testosterone treatment. The longer DHT suppression was apparent in men who received treatment over a mean time of 12 hours compared with those who received treatment over a mean time of 8 hours, the greater decrease in endogenous estradiol levels (Table 2). We are unable to comment on whether DHT and endogenous estradiol levels were also suppressed by treatment over a longer time period since we did not examine the issue. reasons for observed suppression of DHT and endogenous estradiol will require further investigation. These data, combined with data previously published by the same group [7] in a separate study, show that there is clear improvement in serum testosterone levels over a mean treatment period of 12 hours, with higher serum levels following treatment for 7 hours. These data are consistent with the hypothesis that peak of testosterone will occur within a few hours after single dose of flagyl. Whether these androgens will ultimately have an effect on endogenous steroid biosynthetic pathways is less clear and may depend upon the dose of testosterone. In addition to showing that treatment with testosterone causes a decrease in endogenous estradiol levels, we also found that androstenedione levels increased. The fact that androstenedione levels increase after testosterone treatment and that does not cause an increase in aldosterone levels are consistent with the idea that testosterone and androstenedione are potent androgenic anabolic agents and the fact that testosterone increased DHT-binding protein [40]. The reason we found an increase in both testosterone and androstenedione levels is not clear.

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